Using MPRA to determine links between genetic variants and human phenotypes

A team of researchers at Stanford University has found a significant subset of allele-independent regulatory sites that have multiple causal variants in linkage disequilibrium. In their paper published in the journal Science, the group describes how they applied a massively parallel reporter assay (MPRA) to certain parts of the human genome to look for links between genetic variants and certain human phenotypes.
A team of researchers at Stanford University has found a significant subset of allele-independent regulatory sites that have multiple causal variants in linkage disequilibrium. In their paper published in the journal Science, the group describes how they applied a massively parallel reporter assay (MPRA) to certain parts of the human genome to look for links between genetic variants and certain human phenotypes.