AI could transform patient education in eye care, new research shows

From hospital leaflets to spoken answers in dozens of languages, new research from the University of East London (UEL) suggests artificial intelligence could dramatically improve how patients learn about serious eye conditions. A research team led by UEL's Dr. Mohammad Hossein Amirhosseini and Dr. Fatima Kalabi from Queen's Hospital in London, in collaboration with Moorfields Eye Hospital in London, and Inselspital University Hospital of Bern in Switzerland, has developed a multilingual, voice-enabled AI chatbot designed to help people understand retinal detachment—a sight-threatening condition that often requires urgent surgery. The system allows patients to ask questions in natural language and receive clear, clinically grounded answers drawn from trusted medical sources.

Poor diet linked to heart disease, but Australia has seen improvements in the last 30 years

A poor diet remains a leading contributor to ischemic heart disease, according to an analysis from 204 countries over a 30-year period, published in Nature Medicine. The authors estimate that suboptimal diet associated with this disease was responsible for more than 4 million ischemic heart disease related deaths and almost 97 million total disability-adjusted life years were lost in 2023. The findings could improve understanding of how specific diets influence ischemic heart disease patterns and support nutrition-focused strategies to reduce health impacts.

How one ‘forever chemical’ can disrupt a baby’s facial development

Researchers have long associated per- and polyfluoroalkyl substances (PFAS), commonly known as "forever chemicals," with certain severe birth defects, but exactly how these pollutants harm a developing fetus has remained mostly a mystery. New research now provides the first clear molecular explanation, showing how one PFAS, called perfluorodecanoic acid (PFDA), can trigger craniofacial abnormalities before birth. The research was published today in Chemical Research in Toxicology.

Toxic RNA exposure, not repeat growth, may drive worsening DM1 heart disease

Myotonic dystrophy type 1 (DM1) is the most common cause of adult-onset muscular dystrophy, a genetic disorder that leads to muscle weakness and wasting, but also affects the brain, the gastrointestinal tract and the heart. In a study published in the JCI Insight, researchers at Baylor College of Medicine focused on the effects of DM1 in the heart. Their findings help answer questions about why the disease worsens over time and whether the damage can be reversed once it has begun.